Osteoarthritis & Joint Repair
Mechanics, inflammation, immune signaling, and regenerative strategies to rebuild joint stability and motion.
We build human-relevant models of degenerative disease and apply exposure biology, stem cell systems, and translational gene/cell therapy. Our aim is to move from “managing degeneration” to preventing and repairing it.
Monthly updates • Global collaboration welcome • ORCID-verified identity
We integrate stem cell biology, organoid engineering, exposure biology, and gene/cell therapy to design actionable interventions for joint repair and tissue regeneration.
Mechanics, inflammation, immune signaling, and regenerative strategies to rebuild joint stability and motion.
How pollution and toxicants accelerate degeneration; mapping stress and immune activation in joint tissue.
Human joint organoids that mimic cartilage/synovium to observe disease emergence and screen repair strategies.
Targeted delivery and cell-based repair to slow degeneration, control inflammation, and restore function.
Stem-cell-derived joint organoids are exposed to real-world pollutants and controlled mechanical stress. We track degeneration in real time and evaluate regenerative gene/cell therapy strategies intended to repair cartilage rather than only treat pain.
Defined airborne mixtures induce inflammatory signatures linked to aggressive osteoarthritis—now a platform for anti-pollution regenerative testing.
Concept to slow cartilage erosion and promote structural recovery in high-load regions.
Seeking collaborators in early joint degeneration, exposure-driven inflammation, and regenerative delivery strategies.
Briefings cover joint repair models, organoid platforms, and translational strategies for osteoarthritis and exposure-driven tissue damage.
We share organoid disease models, exposure assays, and regenerative repair concepts. If you work on osteoarthritis, environmental toxicology, stem cell engineering, CRISPR, organoid modeling, or translational gene/cell therapy—let’s talk.